Leucemie Linfomi Mielomi

Leucemie Linfomi Mielomi

The Di Bella Method (DBM) improved survival, objective response and performance status in a retrospective observational clinical study on 55 cases of Lymphomas

Neuro Endocrinol Lett. 2012 Dec 28;33(8).
Di Bella G, Colori B, Mascia F.
Observational study on 55 patients affected lymphoma, treated with the Di Bella Method (DBM). The 1, 3 and 5-year survival rates are reported, together with any signs of toxicity. The patients were able to continue the treatment at home, carrying out their normal activities without problems.
Di Bella Method (DBM).

Effects of exogenous melatonin and circadian synchronization on tumor progression in melanoma-bearing C57BL6 mice

J Pineal Res. 2008 Apr;44(3):307-15.
Otálora BB, Madrid JA, Alvarez N, Vicente V, Rol MA.
Importance of taking into account endogenous rhythmicity and limiting melatonin administration to the subjective night (2 mg/kg BW/day) in order to restrict melanoma progression.
Circadian rhythmicity.

sis in B-lymphoma cells

Ann N Y Acad Sci. 2009 Aug;1171:345-9.
Paternoster L, Radogna F, Accorsi A, Cristina Albertini M, Gualandi G, Ghibelli L.
Melatonin is considered a promising antitumor agent, promoting apoptosis in tumor cells and contrasting it in normal cells.
Apoptosis modulator.

Health effects of magnetic fields generated from power lines: new clues for an old puzzle

Ann Ist Super Sanita. 2009;45(3):233-7. Review.
Comba P, Fazzo L.
50-60 Hz magnetic fields as "possibly carcinogenic to humans" because of the "limited evidence" of carcinogenicity of residential exposure relatively to childhood leukemia, perhaps for subtle differences in the timing of melatonin release.
50-60 Hz magnetic fields.

The pineal and regulation of fibrosis: pinealectomy as a model of primary biliary cirrhosis: roles of melatonin and prostaglandins in fibrosis and regulation of T lymphocytes

Med Hypotheses. 1979 Apr;5(4):403-14.
Cunnane SC, Manku MS, Horrobin DF.
Melatonin PGE1 and TXA2 are important in the regulation of fibrosis and defective T suppressor cell function.
Prostaglandin (PG) E1 Thromboxane (TX) A2


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